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Synthetic nootropics & research compounds

SR9009

SR9009 (Stenabolic; Rev-erb agonist)

SR9009, often sold as Stenabolic, is a synthetic Rev-erb agonist and metabolic modulator, not a selective androgen receptor modulator and not a validated nootropic supplement. The public evidence is mostly animal and cell research on circadian rhythm, metabolism, sleep/wake regulation, neural precursor cells, and off-target biology. No reliable human SR9009 dose, onset, safety margin, cognitive benefit, or clinical supplement use was found. In WADA-code sport, the 2026 list names SR9009 among S4.4.1 Rev-erbα agonists that are prohibited at all times.

Hormonal Axis RelatedIsolated CompoundSyntheticMostly Animal EvidenceMostly In Vitro EvidenceEmerging ResearchInsufficient Evidence
Updated August 2026/6 min read/10 citations
Reviewed by the NootropicsIndex editorial team.Method: prioritize cited human research and official safety or regulatory sources; compare retailer-listed prices without endorsing products.Affiliate disclosure
01

What is it?

What SR9009 is

2

SR9009 is a synthetic Rev-erb agonist also sold online as Stenabolic. It is often grouped with SARMs in supplement markets, but mechanistically it is better described as a Rev-erb/circadian-metabolic modulator, not a selective androgen receptor modulator.

Not a validated human nootropic8
No reliable human study was found to support claims for focus, memory, mood, productivity, sleep quality, endurance, or fat loss. The public evidence base is animal, cell, chemical, anti-doping, and market-risk context.

Preclinical evidence dominates

2

The key SR9009 literature examines REV-ERB pharmacology, circadian behavior, metabolism, sleep/wake regulation, neural precursor-cell biology, and analog design in non-human systems. Those findings are useful for mechanism review, but they do not establish consumer outcomes.

02

Names, aliases and forms

Research CodeSR9009
Trade NameStenabolic
Spelling VariantSR 9009
Regulatory NameRev-erb agonist
Chemical Nameethyl 3-[[(4-chlorophenyl)methyl-[(5-nitrothiophen-2-yl)methyl]amino]methyl]pyrrolidine-1-carboxylate
Molecular FormulaC20H24ClN3O4S
Pubchem Cid57394020
InchikeyMMJJNHOIVCGAAP-UHFFFAOYSA-N
03

Mechanisms of action

Rev-erb agonism links circadian and metabolic biology

2

REV-ERB-alpha and REV-ERB-beta are nuclear receptors tied to circadian transcription and metabolism. SR9009 is used in research to probe those pathways, which is why sleep/wake, glucose/lipid, liver, activity, and endocrine-adjacent cautions matter.

Off-target findings weaken simple mechanism claims

5

A PNAS paper reported REV-ERB-independent effects of SR9009 on cell proliferation and metabolism. That does not prove a specific human harm, but it means vendor explanations that present SR9009 as a clean, predictable Rev-erb switch are too simple.

04

Potential effects and evidence map

SR9009 evidence is preclinical. The key Nature paper introduced synthetic REV-ERB agonists including SR9009 and reported effects on circadian behavior and metabolism in experimental models. Later mouse work examined sleep/wake regulation and Rev-erb beta dependence, while cell work reported concentration-dependent neural precursor effects and a PNAS paper warned that SR9009 can have REV-ERB-independent effects on cell proliferation and metabolism. ClinicalTrials.gov searches for SR9009, SR 9009, and Stenabolic returned no human studies suitable for nootropic efficacy, dose, onset, or safety claims.

Effect domainMagnitudeGrade
Focus / attention8
UnknownConfidence: Medium

No reliable human focus or attention benefit has been established for SR9009.

Note Do not infer a human focus effect from circadian, metabolic, animal, or cell studies.

0/5
D
Memory / learning6
UnknownConfidence: Medium

No reliable human memory or learning benefit has been established for SR9009.

Note The neural-cell paper is in vitro and does not validate human cognition.

0/5
D
Mental energy / wakefulness4
UnknownConfidence: Low

Mouse sleep/wake findings do not establish a safe or reliable human wakefulness, energy, or productivity effect.

Note A wake-inducing mouse finding is not a public nootropic timing guide.

1/5
D
Physical energy / endurance7
UnknownConfidence: Low

Exercise and metabolic claims remain preclinical and analog-heavy, with no human SR9009 endurance validation.

Note This contextualizes the marketplace claim without endorsing use.

1/5
D
05

Typical dosages and timing

Dose context8Not established

Do not convert mouse injections, cell concentrations, analog studies, vendor labels, or forum cycles into a human SR9009 dose. No public supplement dose, cycle length, monitoring plan, or safety boundary was found.

06

Timing & effect horizon

Effect horizonUnknown
Unknown
Onset, duration and timing are not established3

Because SR9009 research is tied to circadian biology, timing could matter, but no reliable human onset, duration, The time for blood levels to fall by half during the final elimination phase.Source, best-time, or sleep/wake timing guide was found.

07

Safety, side effects and risk profile

Circadian effects are not automatically useful4
Animal wakefulness or rhythm findings should not be read as safe stimulation, better sleep, or improved productivity. Circadian manipulation can plausibly worsen sleep, mood, alertness, glucose control, or medication timing in some users.
Legal / regulatory risk10
Confidence: High

For WADA-code athletes, SR9009 is among the named S4.4.1 Rev-erbα agonists, prohibited both in and out of competition.

Note USADA also notes SR9009/Stenabolic is a prohibited substance sometimes marketed as a SARM.

5/5
B
Evidence uncertainty8
Confidence: High

No human nootropic efficacy, dose, onset, duration, or long-term safety evidence was found.

Note The strongest evidence is animal/cell mechanism research plus trial-registry absence.

4/5
D
Overstimulation / insomnia4
Confidence: Medium

SR9009 affects sleep/wake biology in animal models, so circadian disruption, insomnia, and wakefulness effects are plausible concerns rather than proven benefits.

Note Human direction, magnitude, and timing are not established.

3/5
D
Interaction risk2
Confidence: Medium

Circadian, metabolic, endocrine, glucose/lipid, sleep, psychiatric, and medication contexts create high uncertainty for unsupervised use.

Note There is no human interaction map for SR9009.

4/5
D
Pregnancy / lactation caution8
Confidence: High

Pregnancy, attempts to conceive, lactation, adolescence, and fertility-sensitive contexts are inappropriate for unsupervised SR9009 exposure.

Note No human reproductive or developmental safety margin was found.

5/5
D
Cardiovascular caution2
Confidence: Low

Human cardiovascular effects are unknown, and a compound that targets circadian-metabolic nuclear-receptor biology should not be assumed cardiovascular-neutral.

Note This is an uncertainty caution, not evidence of a specific human cardiovascular adverse event.

3/5
D
No human safety margin8Unknown

People with sleep disorders, bipolar-spectrum symptoms, anxiety, cardiovascular disease, diabetes, liver disease, endocrine disorders, pregnancy, lactation, fertility concerns, or complex medication regimens should avoid unsupervised SR9009 exposure.

09

Practical buying and quality notes

  • If a listing presents SR9009 as a SARM, fat-loss shortcut, endurance shortcut, or clean stimulant, the label is already blurring basic identity and evidence. Verify independent analytical testing, batch identity, and legal/sport risk before comparing price.9
  • Product rows and price links can document market availability, but they should not be read as a recommendation to buy or use SR9009. The anti-doping, evidence, purity, and safety sections should be read first.10
10

FAQ

Is SR9009 a SARM?

10

No. It is often marketed near SARMs, but SR9009 is a synthetic Rev-erb agonist and metabolic modulator, not a selective androgen receptor modulator.

Is SR9009 a reliable nootropic supplement?

8

No. No human evidence was found for reliable focus, memory, mood, productivity, or healthy-user cognitive benefits.

What SR9009 dose is established?

8

No public human supplement dose is established. Animal, cell, vendor, and forum numbers should not be converted into a self-use plan.

Is SR9009 banned in sport?

10

Yes in WADA-code sport. SR9009 is covered by the S4 category for Rev-erb agonists, which applies at all times.

Why mention off-target effects?

5

Because SR9009 is often sold with simple Rev-erb marketing claims. Published cell work reports REV-ERB-independent effects, so the mechanism should be described as uncertain rather than clean or predictable.

11

References

  1. PubChem. SR9009, CID 57394020. Accessed 2026-06-30.
  2. Solt LA, Wang Y, Banerjee S, et al. Regulation of circadian behaviour and metabolism by synthetic REV-ERB agonists. Nature. 2012;485(7396):62-68.
  3. Banerjee S, Wang Y, Solt LA, et al. Pharmacological Targeting the REV-ERBs in Sleep/Wake Regulation. PLoS One. 2016;11(9):e0162452.
  4. Doi M, Murai I, Kunisue S, et al. REV-ERBbeta is required to maintain normal wakefulness and the wake-inducing effect of dual REV-ERB agonist SR9009. Biochemical Pharmacology. 2018;150:1-8.
  5. Dierickx P, Emmett MJ, Jiang C, et al. SR9009 has REV-ERB-independent effects on cell proliferation and metabolism. Proceedings of the National Academy of Sciences. 2019;116(25):12147-12152.
  6. Morioka N, Abe H, Saeki M, et al. REV-ERB Agonist SR9009 Regulates the Proliferation and Neurite Outgrowth/Suppression of Cultured Rat Adult Hippocampal Neural Stem/Progenitor Cells in a Concentration-Dependent Manner. Cellular and Molecular Neurobiology. 2022;42:1907-1920.
  7. Zhu Y, et al. Regulation of exercise ability and glycolipid metabolism by synthetic SR9009 analogues as new REV-ERB-alpha agonists. Bioorganic & Medicinal Chemistry. 2024;106:117845.
  8. ClinicalTrials.gov search results for SR9009, SR 9009, and Stenabolic. Accessed 2026-06-30.
  9. U.S. Anti-Doping Agency. Selective Androgen Receptor Modulators (SARMs). Accessed 2026-06-30.
  10. World Anti-Doping Agency. The 2026 Prohibited List: International Standard. Version effective 1 January 2026.
Note

This page is informational and summarizes available evidence. It is not medical advice, a recommendation, or a substitute for guidance from a qualified clinician.